HL Deb 24 January 2000 vol 608 cc169-70WA
Baroness Nicol

asked Her Majesty's Government:

Whether there is a relationship between organophosphate pesticides and the illnesses suffered by some veterans of the 1990–91 Gulf conflict in view of the recent reports from the Committee on Toxicity of Chemicals in Food, Consumer Products and the Environment (COT) and the Institute of Occupational Medicine (IOM). [HL663]

Baroness Symons of Vernham Dean

The COT report, published in November, concluded that neuropsychologic al abnormalities can occur as a long-term complication following acute organophosphate (OP) poisoning, particularly if the poisoning is severe. Persistent peripheral neuropathy (disorders of the peripheral nerves,' may occur, but not generally at a level which would give rise to symptoms. The body of evidence gives little support to the hypothesis that low-level exposure to OPs can cause chronic disease of the nervous system. However, the report notes that there remains a question over whether a small proportion of subjects may be at increased risk of clinically significant disease following low level exposure, and recommends further research in this area.

The Institute of Occupational Medicine (IOM) report on the relationship between OP sheep dips and illness in exposed sheep farmers and dippers found the critical exposure factor to be contact with concentrate dip: much higher rates of symptoms, predominantly of a sensory nature, were reported among those who had been principal concentrate handlers. There is very limited evidence that long-term low-level exposure to organophosphates leads to long-term neurotoxic effects.

The conclusions from these reports are in line with the findings of the Defence Scientific Advisory Council's (DSAC) Working Party report, the publication of which we announced on 20 October 1999 and which reviewed existing literature on the long term neurotoxicity of anticholinesterases. It concluded that high doses of organophosphates may have long-term effects on the peripheral nervous system, but that there is limited evidence about long-term toxic effects following low doses.

During the Gulf conflict very few UK service personnel would have handled concentrated OPs. Those that did would have been Environmental Health Officers or technicians, or individual regimental hygiene duties personnel who were appropriately trained in procedures and the use of equipment. There was no evidence of acute organophosphate poisoning or of subsequent unusual ill health arising in this small group.

Neither the IOM nor the COT report provides evidence which would cause us to reassess our view of the possible role of OPs in relation to Gulf Veterans' illnesses. The case for ill-health effects resulting from long term low level exposure to OPs remains unproven and there is currently no reason to believe that Gulf veterans who might have been in casual contact with dilute pesticide (e.g. from the treatment of tents and equipment) or with the malathion dust used to delouse Iraqi prisoners of war, are at increased risk of long term ill-health.

Although it remains the case that there is limited evidence of a link between low-level exposure to OPs and adverse health effects, MoD will continue to monitor the debate about the safety of OPs. We will continue to review scientific evidence in the light of potential exposures which may have occurred in the Gulf and follow future research, including that recommended by the COT report, very closely.